Atlas Architect

Decision Instrument

Risk Intelligence

Failure Modes / Red Flags

Explicitly pressure-test why first-generation AAV may fail for this program, define kill criteria, and map de-risking experiments.

Why first-generation AAV fails here

Target organ underexposed

Severity

Critical

Evidence quality

Moderate

De-risking experiment

Quantitative biodistribution in NHP.

Liver sink too strong

Severity

High

Evidence quality

Moderate

De-risking experiment

Dose-ranging with liver detargeting control arm.

Dose likely too high

Severity

High

Evidence quality

Moderate

De-risking experiment

Efficacy-vs-dose inflection study.

Barrier penetration insufficient

Severity

Critical

Evidence quality

Low

De-risking experiment

In vivo imaging + compartmental sampling.

Pre-existing antibodies block activity

Severity

High

Evidence quality

High

De-risking experiment

Serotype panel neutralization survey.

Innate immune activation

Severity

High

Evidence quality

Moderate

De-risking experiment

Cytokine and complement profiling.

Payload size mismatch

Severity

Medium

Evidence quality

High

De-risking experiment

Payload redesign or split strategy.

Poor manufacturability

Severity

Medium

Evidence quality

Moderate

De-risking experiment

Upstream yield and QC stress run.

Species translation uncertain

Severity

Critical

Evidence quality

Low

De-risking experiment

Cross-species validation ladder.

Post-entry inefficiency

Severity

High

Evidence quality

Low

De-risking experiment

Cellular trafficking mechanistic assay.

Key unknowns & kill criteria

Kill if CNS:liver exposure ratio stays below predefined threshold in NHP.
Kill if neutralization prevalence blocks candidate activity in most baseline sera profiles.
Redesign if innate immune activation exceeds acceptable translational window.
Escalate local-route fallback if systemic barrier penetration remains inconsistent.